oral curcumin+resveratrol !!!!!!
- Έναρξη μίζας kapa
- Ημερομηνία έναρξης
Παρακαλώ συνδεθείτε ή εγγραφείτε για να απενεργοποιήσετε τις διαφημίσεις.
kapa
Μέλος του προσωπικού
Ntoon91 είπε:Μμμ ευχαριστω, για να δοκιμασεις δλδ ενα combo ω3 κουρκουμινη και resv ανεβαινει ενα 250+ euros το χρονο. Τουλαχιστον ευχομαι να τον πιασουμε τον goku στο αβαταρ σΟυρά να μην βγάλουμε μόνο
οχι,πολλα ειναι,με λιγο ψαξιμο αν μιλαμε και για τις 3 ουσιες θα βγει 100 ευρω τον χρονο.
παιζουν μεγαλες αποκλισεις στις τιμες στα συμπληρωματα.
εγω αυτο http://www.ebay.com/itm/Turmeric-Curcumin-Piperine-60-Capsules-500-mg-ANTI-AGING-Bioavailable-Curcumin-/171182142028?pt=LH_DefaultDomain_0&hash=item27db40324c
το πηρα 10 ευρω,μετα εφτασε 38 και τωρα 7.
και ειναι το ιδιο συμπληρωμα απο τον ιδιο πωλητη.
θα βγαλουμε και ουρά και μαλλια
Συνημμένα
tasos
Active Member
μμμμμμ με ανοίξατε την όρεξη ρε παιδιά !
κουρκουμάς ή κιτρινόριζα σαν μπαχαρικό είναι πολύ κοινό
εδώ αναφέρει για τις αντικαρκινικές ιδιότητες του αλλά εχει και συνταγή "το χρυσό γάλα" http://www.terrapapers.com/?p=4886
Φυσική κορτιζόνη:http://parapona-rodou.blogspot.com/2014/01/blog-post_9861.html
Κάπα, και το cayenne έχει παρόμοιες ιδιότητες νομίζω
(ελπίζω να μην χάλασα την συζήτηση)
κουρκουμάς ή κιτρινόριζα σαν μπαχαρικό είναι πολύ κοινό
εδώ αναφέρει για τις αντικαρκινικές ιδιότητες του αλλά εχει και συνταγή "το χρυσό γάλα"
http://www.terrapapers.com/?p=4886Φυσική κορτιζόνη:http://parapona-rodou.blogspot.com/2014/01/blog-post_9861.html
Κάπα, και το cayenne έχει παρόμοιες ιδιότητες νομίζω
(ελπίζω να μην χάλασα την συζήτηση)
kapa
Μέλος του προσωπικού
tasos είπε:μμμμμμ με ανοίξατε την όρεξη ρε παιδιά !
κουρκουμάς ή κιτρινόριζα σαν μπαχαρικό είναι πολύ κοινό
εδώ αναφέρει για τις αντικαρκινικές ιδιότητες του αλλά εχει και συνταγή "το χρυσό γάλα"
Φυσική κορτιζόνη:http://parapona-rodou.blogspot.com/2014/01/blog-post_9861.html
Κάπα, και το cayenne έχει παρόμοιες ιδιότητες νομίζω
(ελπίζω να μην χάλασα την συζήτηση)
η κουρκουμινη ειναι μια αντικαρκινικη ουσια http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2758121/
το cayenne δυστυχως αυξανει τον κινδυνο κατα 15% για καρκινο στομαχου.
ακομα δεν εχω καταλαβει την δοσολογια για το cayenne σαν θεραπεια.
λενε για 5 και 6 καψουλες,πρακτικα αδυνατον
petrosAlex
Member
Καπα να ρωτησω.. Τι ειναι προτιμοτερο ως οραλ.. Φυσικη κουρκουμινη η' σε χαπια (απο εταιρια)
stavros_1983
Member
Να ρωτησω εαν μπλεξουμε epa με κουρκουμινη και ρεσβερατρολη σαν πολλα δεν ειναι η μου φαινεται , και στο κεφαλι καψαικινη, βαλτε και ενα σπιρτο και γιναμε...
kapa
Μέλος του προσωπικού
stavros_1983 είπε:
αν εχεις υπομονη σταδιακα θα τα προσθεσω ολα αυτα.
απο οτι εχω καταλαβει τα Ω δουλευουν σε μεγαλυτερο ποσοστο.
η κουρκουμινη ρεσβερατρολη σε μικροτερο ποσοστο αλλα σε οσους δουλεψε ξεπερασε τα Ω.
ενα κοινο οσων χρησιμοποιησαν κουρκουμινη ρεσβερατρολη ειναι οτι εφυγε η φαγουρα και η ξηροδερμια απο το τριχωτο.
υποψιν οτι υπαρχει και το σεναριο της αποτυχιας.
μην πεσετε σε γιγαντιες ποσοτητες οσοι το δοκιμασετε
kapa
Μέλος του προσωπικού
δειτε αυτο http://www.newsbeast.gr/health/arthro/662834/-resveratroli-ena-fusiko-elixirio-makrozoias/
Η ρεσβερατρόλη, βρίσκεται κυρίως σε υψηλές συγκεντρώσεις στη φλούδα των σταφυλιών και ιδιαίτερα των κόκκινων σταφυλιών, στα φύλλα ευκαλύπτου και ελάτου, στους ξηρούς καρπούς, στα μούρα, και σε μικρότερες ποσότητες έχει βρεθεί σε 70 ακόμη φυτικά προϊόντα. Η περιεκτικότητα στη φλούδα των φρέσκων κόκκινων σταφυλιών σε ρεσβερατρόλη είναι 50-100 μg/g, ενώ στο κόκκινο κρασί μπορεί να βρεθεί σε συγκεντρώσεις 1,5-3 mg/L. Πλέον, έχουν αναπτυχθεί και συμπληρώματα διατροφής που περιέχουν σημαντικές ποσότητες ρεσβερατρόλης.
αλλα δειτε και αυτο http://skepsitrofikinisi.blogspot.gr/2012/10/blog-post_4755.html
Η ρεσβερατρόλη, βρίσκεται κυρίως σε υψηλές συγκεντρώσεις στη φλούδα των σταφυλιών και ιδιαίτερα των κόκκινων σταφυλιών, στα φύλλα ευκαλύπτου και ελάτου, στους ξηρούς καρπούς, στα μούρα, και σε μικρότερες ποσότητες έχει βρεθεί σε 70 ακόμη φυτικά προϊόντα. Η περιεκτικότητα στη φλούδα των φρέσκων κόκκινων σταφυλιών σε ρεσβερατρόλη είναι 50-100 μg/g, ενώ στο κόκκινο κρασί μπορεί να βρεθεί σε συγκεντρώσεις 1,5-3 mg/L. Πλέον, έχουν αναπτυχθεί και συμπληρώματα διατροφής που περιέχουν σημαντικές ποσότητες ρεσβερατρόλης.
αλλα δειτε και αυτο http://skepsitrofikinisi.blogspot.gr/2012/10/blog-post_4755.html
Παρακαλώ συνδεθείτε ή εγγραφείτε για να απενεργοποιήσετε τις διαφημίσεις.
Ένα πολύ καλογραμμένο άρθρο σχετικά με την dht και το οξειδωτικό στρες.
Stop Fearing Testosterone & DHT
Is there really a smarter way to stop hair loss? An approach that does not involve playing around with the androgens, testosterone and its more powerful metabolite? What if there was a way to maximize muscle size, strength, libido, mood and keep your hair at the same time? There is indeed a way to stop this hair miniaturization madness without having to resort to petrochemicals that throw your endocrine system into a loop!
Why Testosterone & DHT are Not your Enemy
Before we delve into this topic, let’s understand first and foremost that testosterone and dihydrotestosterone (DHT) are not the enemy. Before you stop reading, consider for a moment that when testosterone is bound to sex hormone binding globulin (SHBG), it cannot be converted into DHT via the enzyme 5-alpha reductase.
In other words, only free testosterone (unbound) is subject to conversion to DHT by 5-alpha reductase. Research has shown that when hormone parameters are measured in balding vs non-balding men, there is a significant reduction in SHBG levels observed in bald men, compared with non-balding. [1]
Balding Men Typically Have Lower Testosterone & DHT levels
Originally published in 1992, the American Journal of Physical Anthropology examined total testosterone and DHT in blood serum as well as free testosterone in saliva in 110 healthy young men. The absolute serum androgen concentrations in men with a disposition to balding is lower than in men with no alopecia (hair loss).
The popular assumption (myth) that androgen levels are generally elevated in balding men should be rejected. In accordance with this finding, men with a disposition to balding are morphologically no more masculine than those with good scalp hair growth. When body build and age are taken into consideration.
Simply stated, non-balding scalps typically have higher serum (blood) levels of DHT. This is not a misprint. [2]
How Does the Enzyme 5-AR (5-alpha reductase) Fit into this Equation?
Let’s get one thing about of the way first as you might be wondering how the enzyme 5-alpha reductase fits into hair loss. The answer is, yes it does matter. In fact, androgenetic alopecia or hormone mediated hair loss is absolutely correlated with this enzyme. Note that while serum (blood levels) of DHT are inversely correlated with hair loss, salivary DHT is directly correlated.
So to be clear, if you test your serum DHT level, it’s not going to be of any real value. Generally speaking, young men who bald have low levels of SHBG, which allows plenty of unbound testosterone to freely convert via 5-alpha reductase. However, as we age, our SHBG levels naturally increase. As we approach middle-age, these are often too high and we may suffer from a lack of bio-available testosterone. Also with respect to aging, through the process of aromatization (conversion of testosterone into estrogen) as well as increased production of 5-alpha reductase, local tissue expression of these enzymes affects hair and androgen levels alike. [3]
What’s the Real Culprit in Male Pattern Baldness (MPB)?
The simple explanation is inflammation. Inflammation is not always obvious, it can produce a low-grade chronic infection that is not always visible to the eye. Inflammation involves a form of oxygen deprivation, which in the case of hair loss can result from an infection.
When androgens are affecting local tissue, the difference between a balding and non-balding person is that significantly more DHT is necessary to inflame the latter.
However, those who bald are quite sensitive to DHT, and a result generate a high volume of reactive oxygen species (ROS). Think of ROS as free-radicals.
Reactive oxygen species are associated with accelerated aging in cells (senescence). Moreover, ROS by way of oxidative stress (free-radicals) damages cellular DNA, proteins and fatty acids.
If that sounds bad, well it is, and as a result induces transforming growth factor-beta (TGF-β) in hair producing cells called the dermal papilla. [4] It is transforming growth factor-beta that stops the growth of hair follicles. Simply stated, TGF-β is a hair follicle assassin. If it can be tamed, your hair can take less of a “hit” without having to experiment with hormones.
Neutralizing the ROS Effect
When physiological levels of androgen exposure exist to boost reactive oxygen species (ROS) generation in androgen “sensitive” hair follicles, TGF-β1 secretion can be mitigated. How? The very best way is to boost your body’s ability to increase its own antioxidant enzymes. These antioxidant enzymes, such as superoxide dismutase (SOD), glutathione peroxidase, and catalase all work together in human cells against toxic reactive oxygen species. [5] Reactive oxygen species (ROS) are not always bad, they have a purpose. Low concentrations of ROS can be beneficial or even indispensable in processes such as intracellular signaling and defense against micro-organisms. Standard antioxidants are not nearly sufficient to halt the process because they do not generate antioxidant enzymes. However, antioxidant enzyme boosters can. Certain plant and sea-based polyphenols can epigenetically “turn on” gene and/or transcription factors that will boost the level of antioxidant protection within inflamed tissue. These are thousands of times more powerful than the traditional antioxidants.
Additional Information
So let’s review…androgens are involved in ROS generation, and ROS is related to TGF-β secretion. This is one of the significant events that are involved in androgenetic alopecia or male pattern baldness. In tests during sub-cultivation of hair cells (dermal papilla cells), sensitivity to androgens may be low because of the reduced expression level of the andogen receptor. Androgen receptors are expressed at higher levels within the hair cells of bald frontal scalps than those of non-balding occipital scalps.
The most effect antioxidant enzyme boosters for hair growth purposes in my experience are the following: Ecklonia cava, curcumin, and trans-resveratrol.
Επίσης αντίστοιχη δράση και τα πεπτίδια χαλκού αν και σε αρκετά χαμηλότερο βαθμό.
The Effect of Tripeptide Copper Complex on Human Hair Growth
If you’ve been looking into ways to treat hair loss for a while, chances are you have at least heard of “copper peptides” for hair growth. Copper peptides are the mineral copper, bonded with three protein fragments (peptides). These peptides are the amino acids alanine, histidine and lysine. Better known as copper-AHK (Cu-AHK).
The primary mechanisms are many. Its effects on growth on many types of cells, such as boosting the number (proliferation) of fibroblasts, increasing the production of vascular endothelial growth factor (VEGF), which is a substance that allows the formation of new blood vessels. Copper peptides also reduces the secretion of transforming growth factor-beta1 within the fibroblasts. Reducing this secretion is crucial to void out the negative effects of DHT. [1]
The ultimate result is less oxidative stress (free-radical attack) and a stimulation of hair growth by the dermal papilla cells. It is oxidative stress that stimulates elevated DHT formation in scalp follicles. This copper complex inhibits oxidative stress by raising the production of SOD (Superoxide dismutase), which in turn neutralizes the potent free-radical superoxide.
The Other Copper Tripeptide (Cu-GHK)
The peptide Gly-His-Lys (GHK) is a naturally occurring copper found in human serum and cerebrospinal fluid. Copper-GHK plays a physiological role in the process of wound healing and tissue repair by stimulating collagen synthesis in fibroblasts. Moreover, copper-GHK promotes the survival of basal stem cells in the skin. [2] Several studies on aged skin have demonstrated that Cu-GHK tightens skin, improves elasticity and firmness, reduces fine lines, wrinkles, photo-damage and hyperpigmentation. [3]
Όπως είχαμε αναφέρει και παλιότερα σε άλλο θέμα, αυτό που είχε προτείνει ο tropheus δείχνει να είναι ένα πολύ ολοκληρωμένο συμπλήρωμα, αλλά πολύ ακριβό
http://www.ebay.com/itm/Marine-D3-Anti-Aging-Lower-Blood-Pressure-Omega-3-Antioxidant-Seanol-P-DHA-EPA-/281316424093?pt=US_Dietary_Supplements_Nutrition&hash=item417fc3f99d
μαζί με curcumin+resveratrol+τοπικά πεπτίδια χαλκού.
Stop Fearing Testosterone & DHT
Is there really a smarter way to stop hair loss? An approach that does not involve playing around with the androgens, testosterone and its more powerful metabolite? What if there was a way to maximize muscle size, strength, libido, mood and keep your hair at the same time? There is indeed a way to stop this hair miniaturization madness without having to resort to petrochemicals that throw your endocrine system into a loop!
Why Testosterone & DHT are Not your Enemy
Before we delve into this topic, let’s understand first and foremost that testosterone and dihydrotestosterone (DHT) are not the enemy. Before you stop reading, consider for a moment that when testosterone is bound to sex hormone binding globulin (SHBG), it cannot be converted into DHT via the enzyme 5-alpha reductase.
In other words, only free testosterone (unbound) is subject to conversion to DHT by 5-alpha reductase. Research has shown that when hormone parameters are measured in balding vs non-balding men, there is a significant reduction in SHBG levels observed in bald men, compared with non-balding. [1]
Balding Men Typically Have Lower Testosterone & DHT levels
Originally published in 1992, the American Journal of Physical Anthropology examined total testosterone and DHT in blood serum as well as free testosterone in saliva in 110 healthy young men. The absolute serum androgen concentrations in men with a disposition to balding is lower than in men with no alopecia (hair loss).
The popular assumption (myth) that androgen levels are generally elevated in balding men should be rejected. In accordance with this finding, men with a disposition to balding are morphologically no more masculine than those with good scalp hair growth. When body build and age are taken into consideration.
Simply stated, non-balding scalps typically have higher serum (blood) levels of DHT. This is not a misprint. [2]
How Does the Enzyme 5-AR (5-alpha reductase) Fit into this Equation?
Let’s get one thing about of the way first as you might be wondering how the enzyme 5-alpha reductase fits into hair loss. The answer is, yes it does matter. In fact, androgenetic alopecia or hormone mediated hair loss is absolutely correlated with this enzyme. Note that while serum (blood levels) of DHT are inversely correlated with hair loss, salivary DHT is directly correlated.
So to be clear, if you test your serum DHT level, it’s not going to be of any real value. Generally speaking, young men who bald have low levels of SHBG, which allows plenty of unbound testosterone to freely convert via 5-alpha reductase. However, as we age, our SHBG levels naturally increase. As we approach middle-age, these are often too high and we may suffer from a lack of bio-available testosterone. Also with respect to aging, through the process of aromatization (conversion of testosterone into estrogen) as well as increased production of 5-alpha reductase, local tissue expression of these enzymes affects hair and androgen levels alike. [3]
What’s the Real Culprit in Male Pattern Baldness (MPB)?
The simple explanation is inflammation. Inflammation is not always obvious, it can produce a low-grade chronic infection that is not always visible to the eye. Inflammation involves a form of oxygen deprivation, which in the case of hair loss can result from an infection.
When androgens are affecting local tissue, the difference between a balding and non-balding person is that significantly more DHT is necessary to inflame the latter.
However, those who bald are quite sensitive to DHT, and a result generate a high volume of reactive oxygen species (ROS). Think of ROS as free-radicals.
Reactive oxygen species are associated with accelerated aging in cells (senescence). Moreover, ROS by way of oxidative stress (free-radicals) damages cellular DNA, proteins and fatty acids.
If that sounds bad, well it is, and as a result induces transforming growth factor-beta (TGF-β) in hair producing cells called the dermal papilla. [4] It is transforming growth factor-beta that stops the growth of hair follicles. Simply stated, TGF-β is a hair follicle assassin. If it can be tamed, your hair can take less of a “hit” without having to experiment with hormones.
Neutralizing the ROS Effect
When physiological levels of androgen exposure exist to boost reactive oxygen species (ROS) generation in androgen “sensitive” hair follicles, TGF-β1 secretion can be mitigated. How? The very best way is to boost your body’s ability to increase its own antioxidant enzymes. These antioxidant enzymes, such as superoxide dismutase (SOD), glutathione peroxidase, and catalase all work together in human cells against toxic reactive oxygen species. [5] Reactive oxygen species (ROS) are not always bad, they have a purpose. Low concentrations of ROS can be beneficial or even indispensable in processes such as intracellular signaling and defense against micro-organisms. Standard antioxidants are not nearly sufficient to halt the process because they do not generate antioxidant enzymes. However, antioxidant enzyme boosters can. Certain plant and sea-based polyphenols can epigenetically “turn on” gene and/or transcription factors that will boost the level of antioxidant protection within inflamed tissue. These are thousands of times more powerful than the traditional antioxidants.
Additional Information
So let’s review…androgens are involved in ROS generation, and ROS is related to TGF-β secretion. This is one of the significant events that are involved in androgenetic alopecia or male pattern baldness. In tests during sub-cultivation of hair cells (dermal papilla cells), sensitivity to androgens may be low because of the reduced expression level of the andogen receptor. Androgen receptors are expressed at higher levels within the hair cells of bald frontal scalps than those of non-balding occipital scalps.
The most effect antioxidant enzyme boosters for hair growth purposes in my experience are the following: Ecklonia cava, curcumin, and trans-resveratrol.
Επίσης αντίστοιχη δράση και τα πεπτίδια χαλκού αν και σε αρκετά χαμηλότερο βαθμό.
The Effect of Tripeptide Copper Complex on Human Hair Growth
If you’ve been looking into ways to treat hair loss for a while, chances are you have at least heard of “copper peptides” for hair growth. Copper peptides are the mineral copper, bonded with three protein fragments (peptides). These peptides are the amino acids alanine, histidine and lysine. Better known as copper-AHK (Cu-AHK).
The primary mechanisms are many. Its effects on growth on many types of cells, such as boosting the number (proliferation) of fibroblasts, increasing the production of vascular endothelial growth factor (VEGF), which is a substance that allows the formation of new blood vessels. Copper peptides also reduces the secretion of transforming growth factor-beta1 within the fibroblasts. Reducing this secretion is crucial to void out the negative effects of DHT. [1]
The ultimate result is less oxidative stress (free-radical attack) and a stimulation of hair growth by the dermal papilla cells. It is oxidative stress that stimulates elevated DHT formation in scalp follicles. This copper complex inhibits oxidative stress by raising the production of SOD (Superoxide dismutase), which in turn neutralizes the potent free-radical superoxide.
The Other Copper Tripeptide (Cu-GHK)
The peptide Gly-His-Lys (GHK) is a naturally occurring copper found in human serum and cerebrospinal fluid. Copper-GHK plays a physiological role in the process of wound healing and tissue repair by stimulating collagen synthesis in fibroblasts. Moreover, copper-GHK promotes the survival of basal stem cells in the skin. [2] Several studies on aged skin have demonstrated that Cu-GHK tightens skin, improves elasticity and firmness, reduces fine lines, wrinkles, photo-damage and hyperpigmentation. [3]
Όπως είχαμε αναφέρει και παλιότερα σε άλλο θέμα, αυτό που είχε προτείνει ο tropheus δείχνει να είναι ένα πολύ ολοκληρωμένο συμπλήρωμα, αλλά πολύ ακριβό
http://www.ebay.com/itm/Marine-D3-Anti-Aging-Lower-Blood-Pressure-Omega-3-Antioxidant-Seanol-P-DHA-EPA-/281316424093?pt=US_Dietary_Supplements_Nutrition&hash=item417fc3f99d
μαζί με curcumin+resveratrol+τοπικά πεπτίδια χαλκού.
stavros_1983
Member
Ερωτησεις.....
πρωτον ενα ρεζουμε της παραπανω υποθεσης , δευτερον πως μπορειτε ρε παιδια να εμπιστευτειτε ενα προιον σαν το παραπανω εννοωντας θα ειναι γνησιο η οχι δλδ αγοραζουμε κατι λεμε ουαου το πηρα ειναι αυτο που λεει η οχι μπορεις να το καταλαβεις η απλα δοκιμαζεις και τριτον ασχετο με το θεμα υπαρχει κατι θετικο για apple procyanidins; το ξερω ειναι αλλο θεμα αλλα νομιζω οτι κολλαει με τα αλλα δυο .
πρωτον ενα ρεζουμε της παραπανω υποθεσης , δευτερον πως μπορειτε ρε παιδια να εμπιστευτειτε ενα προιον σαν το παραπανω εννοωντας θα ειναι γνησιο η οχι δλδ αγοραζουμε κατι λεμε ουαου το πηρα ειναι αυτο που λεει η οχι μπορεις να το καταλαβεις η απλα δοκιμαζεις και τριτον ασχετο με το θεμα υπαρχει κατι θετικο για apple procyanidins; το ξερω ειναι αλλο θεμα αλλα νομιζω οτι κολλαει με τα αλλα δυο .
kapa
Μέλος του προσωπικού
stavros_1983 είπε:
θα προσπαθησω να απαντησω σε ολα.
το ρεζουμε των παραπανω ειναι ενα σχημα θεραπειας Ω3,κουρκουμινη,ρεσβερατρολη και τοπικα πεπτιδια χαλκου.
στο δευτερο ερωτημα ειναι οτι δεν εχει υπαρξει ακομα ενα προιον που να μην εχει αυτα που αναφερει.
στο τριτο το αφηνω προς το παρον επειδη δεν εχουμε αναφορες απο χρηστη.
το δοκιμασε στο παρελθον τοπικα καποιος αλλα δεν μας ειπε αποτελεσματα
για τα Ω σε συμπληρωμα και τα πεπτιδια τοπικα επαυξανω.
μενει να δω και στην πραξη τα αλλα δυο
stavros_1983
Member
kapa και τα τρια μαζι δλδ ξυπνας το πρωι παιρνεις ω καπακι κουρκουμινη και καπακι ρεσβερατρολη και το βραδυ κοπανας και χαλκο στο κεφαλι η παιρνεις 1 μηνα ω μετα 1 μηνα κουρ και ρεσβε αν μπορεις διευκρινισε , ευχαριστω .
ρε soltareno τοσο πολυ και γω που νομιζα οτι κατι θα κανει γιατι απο αυτα που διαβασα λιγο μου γεμιζε το ματι .
ρε soltareno τοσο πολυ και γω που νομιζα οτι κατι θα κανει γιατι απο αυτα που διαβασα λιγο μου γεμιζε το ματι .
kapa
Μέλος του προσωπικού
stavros_1983 είπε:
προς το παρον παιρνω Ω,κουρκουμινη και στους κροταφους μονο βαζω airol folligen σε αδειο σημειο.
οτι μεταβολες σε τριχοπτωση εχω δηλαδη ειναι απο συμπληρωματα και μονο και οχι απο τα πεπτιδια χαλκου.
τα πεπτιδια χαλκου τοπικα δινουν λιγο regrowth.
αργοτερα θα βαλω και ρεσβερατρολη σε συμπληρωμα η grape seed.
προς το παρον ειμαι ευχαριστημενος απο τα Ω σε συμπληρωμα και τα τοπικα πεπτιδια σε αποδοση
